viernes, 12 de abril de 2019

Transcranial direct current stimulation and motor rehabilitation: a timely opportunity to boost recovery of brain lesions - On Medicine

Transcranial direct current stimulation and motor rehabilitation: a timely opportunity to boost recovery of brain lesions - On Medicine

Jordi Gandini, Hiroshi Mitoma, Shinji Kakei & Mario Manto

Dr Jordi Gandini is MD, training in the Department of Neurology of the CHU-Charleroi, Belgium.

Professor Hiroshi Mitoma is Professor at the Medical Education Promotion Center, Tokyo Medical University, Japan.

Professor Shinji Kakei is Professor at the Tokyo Metropolitan Institute of Medical Science, Japan.

Professor Mario Manto is Professor at the University of Mons, Belgium.


Transcranial direct current stimulation and motor rehabilitation: a timely opportunity to boost recovery of brain lesions

On the occasion of the Brain Awareness Week, we invited Journal of NeuroEngineering and Rehabilitation’s Deputy Editor Dr. Mario Manto and colleagues to tell us about transcranial direct current stimulation (tDCS), the focus of a new thematic series, and its promising role in motor rehabilitation.
The human, social and economic impact of brain disorders (BDs) is huge. BDs impair quality of life, cause a substantial loss of productivity and resource utilization with high economic costs in every country of the world. The majority of the socioeconomic burden of BDs is caused by the years of active life lost because of the disability. For instance, the societal cost amounted to €84 billion in Spain in 2010. In UK, 45 million patients suffered from BDs with an annual cost of €134 billion.
A key-question for the scientific community is how to promote motor recovery in BDs. Researchers have been trying to identify methods to boost recovery for  decades; through this, several mechanisms slowing or impairing recovery have now been deciphered. For instance, the intact motor cortex contralateral to a mild chronic stroke will drive higher inhibitory volleys, so that the damaged motor cortex will be over-inhibited. This maladaptive plasticity is a source of poor motor recovery.

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