Overexpression of UBQLN1 reduces neuropathology in the P497S UBQLN2 mouse model of ALS/FTD | Acta Neuropathologica Communications | Full Text

Overexpression of UBQLN1 reduces neuropathology in the P497S UBQLN2 mouse model of ALS/FTD | Acta Neuropathologica Communications | Full Text

Overexpression of UBQLN1 reduces neuropathology in the P497S UBQLN2 mouse model of ALS/FTD

Missense mutations in UBQLN2 cause X-linked dominant inheritance of amyotrophic lateral sclerosis with frontotemporal dementia (ALS/FTD). UBQLN2 belongs to a family of four highly homologous proteins expressed in...

Authors:Shaoteng Wang, Micaela Tatman and Mervyn J. Monteiro

Citation:Acta Neuropathologica Communications 2020 8:164

Content type:Research

 

Published on: 7 October 2020

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