Enhanced accumulation of N-terminally truncated Aβ with and without pyroglutamate-11 modification in parvalbumin-expressing GABAergic neurons in idiopathic and dup15q11.2-q13 autism | Acta Neuropathologica Communications | Full Text

Enhanced accumulation of N-terminally truncated Aβ with and without pyroglutamate-11 modification in parvalbumin-expressing GABAergic neurons in idiopathic and dup15q11.2-q13 autism | Acta Neuropathologica Communications | Full Text

Enhanced accumulation of N-terminally truncated Aβ with and without pyroglutamate-11 modification in parvalbumin-expressing GABAergic neurons in idiopathic and dup15q11.2-q13 autism

Autism, the most frequent neurodevelopmental disorder of a very complex etiopathology, is associated with dysregulation of cellular homeostatic mechanisms, including processing of amyloid-β precursor protein (...

Authors:Janusz Frackowiak, Bozena Mazur-Kolecka, Pankaj Mehta and Jerzy Wegiel

Citation:Acta Neuropathologica Communications 2020 8:58

Content type:Research

Published on: 28 April 2020

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